this post was submitted on 19 Jul 2025
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Conclusion:

We confirm from the review of the literature on epidemiological data, meta-analysis, and clinical interventions where dietary cholesterol challenges were utilized that there is not a direct correlation between cholesterol intake and blood cholesterol. This lack of correlation is mainly due to the compensatory mechanisms exerted by the organism to manage excess dietary cholesterol, including decreases in cholesterol absorption and down-regulation of cholesterol synthesis. A great number of epidemiological studies and meta-analysis indicate that dietary cholesterol is not associated with CVD risk nor with elevated plasma cholesterol concentrations. Clinical interventions in the last 20 years demonstrate that challenges with dietary cholesterol do not increase the biomarkers associated with heart disease risk. Further, in the specific circumstances where eggs are the source of dietary cholesterol, an improvement in dyslipidemias is observed due to the formation of less atherogenic lipoproteins and changes in HDL associated with a more efficient reverse cholesterol transport. However, if the cholesterol sources are consumed with saturated and trans fats, as happens in the Western diet pattern, increases in plasma cholesterol may be observed. The most recent epidemiological data and clinical interventions for the most part continue to support the USDA 2015 dietary guidelines that removed the upper limit of dietary cholesterol.

The document reviews the relationship between dietary and blood cholesterol, highlighting that recent epidemiological studies and clinical interventions have found no direct correlation between the two. It notes that outdated dietary guidelines limited cholesterol intake to 300 mg/day, but newer research suggests that dietary cholesterol does not significantly impact blood cholesterol levels.

The review discusses how dietary cholesterol from eggs does not increase the risk of cardiovascular disease (CVD) and may even have beneficial effects on plasma lipoprotein subfractions and HDL cholesterol. It also mentions that the body has mechanisms to manage excess dietary cholesterol, such as decreased absorption and down-regulation of cholesterol synthesis.

The document concludes that current evidence supports the removal of upper limits on dietary cholesterol, as recommended by the 2015 USDA dietary guidelines.

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[–] xep@fedia.io 2 points 6 days ago (1 children)

Unlike The Diabetes/Cancer/Obesity code, a lot of it is anecdotal. Most of the science is in the last third of the book for some reason. You could skip more than half and still get most of the important takeaways from the book! I still found it useful since I've not done any fasts longer than 16 hours before.

[–] psud@aussie.zone 2 points 1 day ago (1 children)

16 hours

The key is to eat too much. I try to just eat pemmican (I should post my recipe again now we've moved twice) my pemmican is just dried meat and rendered suet.

The recent batch was thicker than usual, so when I cut it up each piece was maybe 30% larger than normal

I put a piece on a plate, ate maybe two thirds of it and was full. But I left it in front of me, so over the next hour I picked at it until I had eaten it all.

I didn't eat anything the next day

[–] xep@fedia.io 2 points 1 day ago (2 children)

I would love a recipe for pemmican. Looking forward to it!